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spacerTrabajos Publicados por Imanol Arozarena Martinicorena
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  1. Matallanas, D.; Sanz-Moreno, V.; Arozarena, I.; Calvo, F.; Agudo-Ibáñez, L.; Santos, E.; Berciano, M.T.; Crespo, P. "Distinct utilization of effectors and biological outcomes resulting from site-specific Ras activation: Ras functions in lipid rafts and Golgi complex are dispensable for proliferation and transformation.". Mol. Cell. Biol.. 26(1): 100-116. (2006). (PMID: 16354683).
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  2. Arozarena, I.; Matallanas, D.; Berciano, M.T.; Sanz-Moreno, V.; Calvo, F.; Muñoz, M.T.; Egea, G.; Lafarga, M.; Crespo, P. "Activation of H-Ras in the endoplasmic reticulum by the RasGRF family guanine nucleotide exchange factors.". Mol. Cell. Biol.. 24(4): 1516-1530. (2004). (PMID: 14749369).
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  3. Matallanas, D.; Arozarena, I.; Berciano, M.T.; Aaronson, D.S.; Pellicer, A.; Lafarga, M.; Crespo, P. "Differences on the inhibitory specificities of H-Ras, K-Ras, and N-Ras (N17) dominant negative mutants are related to their membrane microlocalization.". J. Biol. Chem.. 278(7): 4572-4581. (2003). (PMID: 12458225).
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  4. Arozarena, I.; Matallanas, D.; Crespo, P. "Maintenance of CDC42 GDP-bound state by Rho-GDI inhibits MAP kinase activation by the exchange factor Ras-GRF. evidence for Ras-GRF function being inhibited by Cdc42-GDP but unaffected by CDC42-GTP.". J. Biol. Chem.. 276(24): 21878-21884. (2001). (PMID: 11285260).
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  5. Arozarena, I.; Aaronson, D.S.; Matallanas, D.; Sanz, V.; Ajenjo, N.; Tenbaum, S.P.; Teramoto, H.; Ighishi, T.; Zabala, J.C.; Gutkind, J.S.; Crespo, P. "The Rho family GTPase Cdc42 regulates the activation of Ras/MAP kinase by the exchange factor Ras-GRF.". J. Biol. Chem.. 275(34): 26441-26448. (2000). (PMID: 10840034).
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  6. Sanz, V.; Arozarena, I.; Crespo, P. "Distinct carboxy-termini confer divergent characteristics to the mitogen-activated protein kinase p38alpha and its splice isoform Mxi2.". FEBS Lett.. 474(2-3): 169-174. (2000). (PMID: 10838079).
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  7. Delgado, M.D.; Vaqué, J.P.; Arozarena, I.; López-Ilasaca, M.A.; Martínez, C.; Crespo, P.; León, J. "H-, K- and N-Ras inhibit myeloid leukemia cell proliferation by a p21WAF1-dependent mechanism.". Oncogene. 19(6): 783-790. (2000). (PMID: 10698496).
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Instituto de Investigaciones Biomédicas "Alberto Sols"
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